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<Return to Disease List
Cerebellar Ischemic Stroke
  • Description
  • Signalment
  • Clinical Features
  • Neurolocalization
  • Genetics
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Ischemic damage, regardless of cause, results in endothelial cell damage predisposing to thrombosis, necrosis, and hemorrhage. One of the most devastating effects of severe intracranial vascular disease is intracranial hemorrhage. Hemorrhage into and around the brain can result in an associated inflammatory reaction, increases in overall intracranial volume, and increases in intracranial pressure. If bleeding is substantial, hematomas may subsequently form.
Age of Onset: 8-9 years of age
Sex Predisposition: Any sex of animal can be affected
Clinical Course:
Clinical signs may stabilize within the first 48 hours or progress rapidly. Most dogs will improve over the course of 1-10 days.
Dogs may have a history of previous episodes that resolved rapidly, suggestive of a more transient ischemic event.
Clinical Signs:
Posture and Appearance
Periodic opisthotonus
Wide-base stance

Movement
Hypermetria/Dysmetria
Ataxia
Intention tremor
Inability to stand or maintain balance
Unable to remain in a sternal position
Mild spasticity
“lurching” or forward-falling movements
Loss of muscle tone

Proprioception  
Proprioceptive deficits

Cranial Nerves  
Absent menace response
Nystagmus

Spinal Reflexes
Exaggerated spinal reflexes
Rostral and tentorial part of the cerebellum
Unknown
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References
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