Cerebellar Ischemic Stroke
Ischemic damage, regardless of cause, results in endothelial cell damage predisposing to thrombosis, necrosis, and hemorrhage. One of the most devastating effects of severe intracranial vascular disease is intracranial hemorrhage. Hemorrhage into and around the brain can result in an associated inflammatory reaction, increases in overall intracranial volume, and increases in intracranial pressure. If bleeding is substantial, hematomas may subsequently form.
Age of Onset: 8-9 years of age
Sex Predisposition: Any sex of animal can be affected
Clinical Course:
Clinical signs may stabilize within the first 48 hours or progressive rapidly
Clinical signs may stabilize within the first 48 hours or progressive rapidly
Clinical Signs:
Posture and Appearance
Nystagmus
Movement
Ataxia with or without hypermetria
Head tilt
Non-ambulatory
Proprioception
Reduced postural deficits
Proprioceptive deficits
Cranial Nerves
Decreased menace response
Other
May be associated with related vestibular abnormalities
Posture and Appearance
Nystagmus
Movement
Ataxia with or without hypermetria
Head tilt
Non-ambulatory
Proprioception
Reduced postural deficits
Proprioceptive deficits
Cranial Nerves
Decreased menace response
Other
May be associated with related vestibular abnormalities
Rostral and tentorial part of the cerebellum
Unknown
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